Colic-Associated Dystemperament has long been attributed to an emotional reaction to chronic pain rather than what this author believes is responsible: A direct effect of a nerve signal similar to pain that has singular effects upon neural function which, although it is often sent alongside pain signals, may be conveyed to the brain even in the absence of pain signals. While it may be possible that this phenomenon can be explained by the brain gradually changing the way it handles pain signals, I would posit that the pain signals themselves morph with time due to chemical exhaustion of the mitochondria of the cells that make the electrically-active reflector molecules that, when released, cause electrical signals to be bounced back to the brain, leading to the perception of pain. Metabolic exhaustion of the nerve cells in the case of chronic pain result in shortcuts being taken in their manufacturing process, leading to structural changes to the "reflector molecules" resulting in a return signal that is qualitatively different than a traditional pain signal. These signals electrically cue one to become instantly enraged regardless of the actual duration of the pain. Pain signals originating from these "deformed reflectors," rather than having the effects associated with pain e.g. capturing attention and distracting focus away from other tasks, crying for help, and disrupting motor function to prevent further injury, instead provoke extreme anger.